Super Soldiers

Nazi methamphetamines, Ritalin, Adderall - Ready for War

How do our rulers get us to kill one another in war after war, benefiting only these select few kings, queens, oligarchs or elites? How do the basic species-self-preservation instincts and moral dogmas driven into us since birth get subverted. Elementary, my dear Watson, with drugs. Safe and effective drugs to boot (eyes rolling so far back in my head that I saw last week).

Our current generation of super soldiers are primed and ready for video-game-like control of the drones about to take over the world. Shoved full of Ritalin since youth and told they are ADHD and incapable of structured thought without taking one of the most deadly drugs of the last 100 years. Recently re-watched the movie ‘Limitless’ with Bradley (CIA honeypot) Cooper - all true: cannot move up in the SS unless you are focused and perfectly psychotic.

Nazis called them the ‘pep pills’, ‘Panzerschokolade’ (tank chocolate), ‘Luftwaffeschokolade’ (pilot’s chocolate) or were given to SS officers as salts to be added to all meals. Pervitin tablets were given to the German soldiers (and other armies) well into the 1970’s. You cannot rule a globe and murder entire nations unless you are high and destabilized, but focused all at the same time. Zombies of war.

While officially ‘banned’ in the 1970’s, they have continued to be mass produced as one drug or another, for some fictional ailment or another. Today, they are sold under trade names such as Desoxyn but are still all methamphetamine salts. You would think by now, that we would know meth was bad for us.

“Methamphetamine is a member of the class of amphetamines in which the amino group of (S)-amphetamine carries a methyl substituent. It has a role as a neurotoxin, a psychotropic drug, a central nervous system stimulant, a xenobiotic and an environmental contaminant” - https://pubchem.ncbi.nlm.nih.gov/compound/methamphetamine

The next trick the nazi drug companies pulled was to subvert the scientific “process” by slightly altering chemical precursors to create new patents which were then sent through the FDA corrupt approval-jungle (wash, rinse, repeat, ad nauseum). For instance, Adderall is created from a precursor called phenyl-2-nitropropene, where modern, illegal meth drugs are created from the precursor phenyl-2-propanone. You do not need to be a chemist to see the pharma loop-hole used to continually fuel, both legally and illegally, our societal addiction to amphetamines. Keep those super soldiers on ready for the next fascist uprising!

Precursor to illegal Meth - Phenylacetone, also known as phenyl-2-propanone

Precursor to Adderall - phenyl-2-nitropropene

Adderall

Illegal Methamphetamine

Other trade brand names currently include: Adzenys XR-ODT, Evekeo, Dyanavel XR, Adzenys ER, Evekeo ODT, Adderall XR, Mydayis etc.

Going through the FDA approval notes on EVERY amphetamine derived drug currently on the market, they all share the common warnings on the product labels (eg. Adzenys):

“5. WARNINGS AND PRECAUTIONS

5.1 Potential for Abuse and Dependence

CNS stimulants, including ADZENYS XR-ODT, other amphetamine-containing products, and methylphenidate, have a high potential for abuse and dependence.

5.2 Serious Cardiovascular Reactions

Sudden death, stroke, and myocardial infarction have been reported in adults with CNS stimulant treatment at recommended doses. Sudden death has been reported in pediatric patients with structural cardiac abnormalities and other serious heart problems taking CNS stimulants at recommended doses for ADHD.

5.3 Blood Pressure and Heart Rate Increases

CNS stimulants cause an increase in blood pressure (mean increase about 2-4 mm Hg) and heart rate (mean increase about 3-6 bpm).

5.4 Psychiatric Adverse Reactions

Exacerbation Pre-Existing Psychosis CNS stimulants may exacerbate symptoms of behavior disturbance and thought disorder in patients with a pre-existing psychotic disorder. Induction of a Manic Episode in Patients with Bipolar Illness CNS stimulants may induce a mixed or manic episode in patients with bipolar disorder. Prior to initiating treatment, screen patients for risk factors for developing a manic episode (e.g., comorbid or has a history of depressive symptoms or a family history of suicide, bipolar disorder, and depression).

New Psychotic or Manic Symptoms CNS stimulants, at recommended doses, may cause psychotic or manic symptoms, e.g., hallucinations, delusional thinking, or mania in patients without prior history of psychotic illness or mania. If such symptoms occur, consider discontinuing ADZENYS XR-ODT. In a pooled analysis of multiple short-term, placebo-controlled studies of CNS stimulants, psychotic or manic symptoms occurred in 0.1% of CNS stimulant-treated patients compared to 0% in placebo-treated patients.

5.5 Long-Term Suppression of Growth

CNS stimulants have been associated with weight loss and slowing of growth rate in pediatric patients.

5.6 Peripheral Vasculopathy, including Raynaud’s Phenomenon

Stimulants, including ADZENYS XR-ODT, used to treat ADHD are associated with peripheral vasculopathy, including Raynaud’s phenomenon. Signs and symptoms are usually intermittent and mild; however, very rare sequelae include digital ulceration and/or soft tissue breakdown. Effects of peripheral vasculopathy, including Raynaud’s phenomenon, were observed in postmarketing reports at different times and at therapeutic doses in all age groups throughout the course of treatment.”

So the nazi medicines seem to be working just as planned. We have a group of zombified killers that have been desensitized to killing other humans since they were young. But we cannot send these super soldiers back into society, so they are all designed with a shortened life kill switch.

Mechanisms for a Meth-shortened Life

Let us begin with the smallest mechanisms known that amphetamine use disrupts. In an earlier post, it was described how the lab-inserted glycoprotein 120 in the spike protein disrupts microRNA processes including dysregulation of Dicer and Drosha. While not exactly a page-turner, the gist of the post was to show that even minute disruptions on this nuclear level have catastrophic consequences to several cellular-based and life-essential functions.

For instance, disruption via ANY oxidative stress (which amphetamines accomplish by several methods) can cause cardiomyocytes to form via the un-controlled expression and packaging of miRNA that would have been normally ‘cut into pieces’ via the Dicer/Drosha process. Imagine this disruption happening everywhere, but especially the brain, in amphetamine usage. Figure below from ‘Got Myocarditis’ post explains more deeply this dysregulation of the Nrft2 system.

from Got Myocarditis previous post

Research regarding cardiovascular disruption via methamphetamines - Cardiac complications (“Methamphetamine‐related cardiovascular diseases” - summarized)

3.1. Histological findings

3.2. Acute cardiovascular toxicity

3.3. Hypertension related complications

3.4. Heart failure

3.5. Takotsubo cardiomyopathy

3.6. Sudden cardiac death

3.7. Coronary artery disease and myocardial infarction

3.8. Stroke

Central Nervous System Kill Switch

We have known for quite a while that Amphetamines inhibited central nervous system neurons. From a 1979 research paper: “These results suggest that the amphetamine-induced inhibition of NA (noradrenergic) neurons in the LC (locus coeruleus) is an indirect effect, mediated via activation of central α-receptors of presynaptic character.”

So what exactly are noradrenergic neurons and so what if they are inhibited?

“The central noradrenergic system plays crucial roles in maintaining the homeostasis of the adult brain and influences various neural networks that control cognition and behavior. The main source of noradrenaline (NA) in the human brain is the locus coeruleus (LC), a pontine nucleus that supplies NA to the entire central nervous system (CNS), except for the basal ganglia.”

“The LC-NA system modulates many neural networks (macroscale level) and regulates several homeostatic mechanisms (microscale level) of the central nervous system, many of which are involved in neurodevelopmental disorders. At the microscale level, LC-NA participates in synaptogenesis and synaptic plasticity, both as a neurotransmitter and through modulation of microglia and the promotion of blood–brain barrier integrity. At the same time, NA functions as a regulator of neurogenesis, a role that this system maintains during both pre-natal and post-natal life, up to adulthood. At the macroscale level, the LC-NA system regulates the activity of many neural networks, participating in the sleep/wake cycle and stress response. Moreover, it modulates cognitive functions such as attention and memory. Altered synaptogenesis and abnormal neurogenesis are suggested as possible pathogenetic mechanisms of neurodevelopmental disorders; in parallel, cognitive and behavioral alterations and mood and sleep disorders are common clinical features in these patients.” - The Central Noradrenergic System in Neurodevelopmental Disorders: Merging Experimental and Clinical Evidence

Turn on that Central Nervous System fault

Once the meth gets into the locus coeruleus (LC), the entire central nervous system is vulnerable for any attack. For instance, the unprotected CNS can be attacked anytime via electromagnetic frequency bursts, from let’s say - a 5G tower or satellite. “[W]e systematically demonstrate how the complex effects of EMFs in neuronal ion channels are exerted at multiple levels and how their significance in the alteration of neuronal functions is strictly dependent on different parameters relative to the type of field used and the studied cell or tissue.” - Effects of electromagnetic fields on neuronal ion channels: a systematic review

“Meth results in disruption of BBB [blood brain barrier] structure and function and the mechanisms that contribute to BBB disruption are similar to those that are responsible for monoaminergic terminal damage after Meth… [T]here is clear evidence that the acute effects of Meth, including hyperthermia, oxidative stress, excitotoxicity and mitochondrial dysfunction play a role in Meth-induced monoamine terminal damage. Research has focused primarily on the dopaminergic and serotonergic systems because of the direct pharmacological actions of Meth at DAT and SERT. However, the mechanisms responsible for monoaminergic damage are not specific for only neurons and likely cause damage to other cells in the brain. One class of cells that can be damaged by these mechanisms is those that which comprise the BBB.” - Methamphetamine effects on blood-brain barrier structure and function

“METH produces (i) a disruption to physiological DA and NE vesicular storage, by interfering with the vesicular monoamine transporter type-2 (VMAT-2), (ii) it impairs the membrane DA transporter (DAT) [74] and NET [75], (iii) it competitively inhibits intracellular DA and NE metabolism by monoamine oxidase (MAO)-A [76] which are placed within both DA and NE terminals [77]” - The Effects of Locus Coeruleus and Norepinephrine in Methamphetamine Toxicity

Genetic Addiction and Targeting - Find Those Best Soldiers

Imagine knowing who your perfect slaves will be, in a group of humans. Imagine being able to know who will respond to your meth poisoning to become an actual unfeeling-uncaring-sociopath-soldier. Imagine the military industrial complex licking their lips to find the most difficult resource in the world: someone to pull the ‘trigger’ on the weapons created to annihilate mankind. Imagine if AncestryDNA, 23andME and other genetic repositories could look at all the DNA human idiots sent in and develop better precursors to target genetic predisposition to becoming an addicted-super-soldier. Whoops.

Getting back to my boring miRNA, Drosha & Dicer post again, miRNA dysregulation of many systems and the disruption from meth usage works on this microRNA level: “Drugs of abuse modulate gene expression, and produce their rewarding effects of euphoria or pleasure through an interaction with the mesolimbic dopaminergic system, leading to persistent alterations (neuroplastic, structural and functional) in the reward-related and memory-related brain centers. An overview of the pathways involved in miRNA regulation of gene expression in addiction is shown in Figure 1 (below - sorry, I love highlighting)” - New insights into the roles of microRNAs in drug addiction and neuroplasticity

“These findings indicate that DNA methylation alterations may occur in genes of dopaminergic system as well as in AKT1 which interacts with this system. Our studies also found that the promoter DNA hypomethylation of these genes (as the result of methamphetamine abuse) exhibit a corresponding increase in the expression of the affected genes… we found DNA hypomethylation of MB-COMT gene promoter in individuals with methamphetamine psychosis. While dopamine, as one of the most important neurotransmitters is released as a result of internal/external stimuli and affects different types of dopamine receptors, COMT enzyme (more specifically MB-COMT) degrades dopamine in the synaptic cleft. Thus, DNA hypomethylation of MB-COMT gene promoter, which leads to increased gene expression could enhance dopamine degradation in the synaptic cleft” - Methamphetamine-Induced Psychosis Is Associated With DNA Hypomethylation and Increased Expression of AKT1 and Key Dopaminergic Genes

Meth summary: cannot sleep, reduced appetite, increased aggression, increased compulsion, 10 fold increase in chance of psychosis, dysregulation of miRNA functionality necessary for life, 10 fold increase in cardiovascular failure, increase chance of amyloidosis… “quick, we need 10mgs more of methamphetamine to help this child think” - most idiot medical doctors. Medical doctors are nazi war recruiters - prove me wrong.

Ritalin (methylphenidate) - Get those future SS kids in line

PS: you still trust doctors trained by satanists, hey? Then shove that ritalin into your future-boot-stomping-fascist children and hope they do not stomp your head when the color revolution comes. Parents: if you gave your kids medical-doctor prescribed Ritalin already, you created a future-meth-addict and potential psychopath. You did it - pharma (the devil) wins again and your place in hell is awaiting. Yep, you tied that millstone to your necks, alright.

“But if anyone causes one of these little ones who believe in Me to stumble, it would be better for him to have a large millstone hung around his neck and to be drowned in the depths of the sea.” - Jesus Christ - Matthew 18:6

Ritalin - Methylphenidate preparation elucidated

“Patients: 234 consecutive children aged 5 through 15 years who met the Diagnostic and Statistical Manual of Mental Disorders (3rd ed, revised) criteria for ADHD were enrolled. Of these children, 206 had sufficient side effects data for analysis.”

“Changes in brain serotonin level may cause different biological and behavioral changes [14, 15]. SERT is the main factor to regulate brain serotonin concentration [14, 15]. SERT density may have profound effects on frontal cortex activity [13-15]. Serotonergic system is involved both in mood regulation and neuropathology of its disorders [15]. Specific mechanisms of actions of anti-depressants are mediated via modulation of serotonergic system [14]. Some anti-depressants such as SERT inhibitors may antagonize SERT and promote its internalization and/or increase SERT gene expression, which is referred as neuroadaptation [24].”

“According to the findings, Ritalin with the high dose of 20 mg/kg could remarkably enhance the levels of bax and caspase-8 genes compared with those in the control group (p < 0.05). It should be noted that treatment with Ritalin could significantly increase TNF-α and IL-1β levels in isolated cerebellar cells (p < 0.05). Moreover, 20 mg/kg of Ritalin decreased mean volumes of granular layer, white matter, as well as molecular layers. It also reduced the number of Purkinje cells compared with those in control rats. In addition, lower coordination movement was observed in the group receiving Ritalin.”

“Pre-clinical findings indicate that MPH treatment during early development may cause enduring effects on behavioral and brain processes under a variety of conditions, although the functional significance of these findings is largely unknown. Currently, noninvasive methods for use in human subjects are insufficiently sensitive to verify neurobiological evidence derived from pre-clinical research.”